Red hair may be increasing as study points to surprising evolution trend

A study from Harvard Medical School indicates natural selection has favored the red hair gene, resulting in a potential increase in the number of redheaded people as humanity continues to evolve.

By analyzing nearly 16,000 ancient genomes spanning 10,000 years, researchers identified a list of traits that nature is actively pushing forward. Among the most prominent were the genetic variants for red hair.

"Perhaps having red hair was beneficial 4,000 years ago, or perhaps it came along for the ride with a more important trait," the authors noted.

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The study, published in the journal Nature, relied on a large database of ancient DNA from West Eurasia. Using new computing methods, the team was able to filter out random fluctuations in DNA to identify what it called "directional selection."

Directional selection happens when a particular version of a gene gives an organism a strong survival or reproductive advantage, causing it to become more common in a population faster than it would by chance, according to experts.

Prior to this study, scientists only knew of about 21 such instances in human history, one of which was lactose tolerance. This new research uncovered hundreds more.

"With these new techniques and a large amount of ancient genomic data, we can now watch how selection shaped biology in real time," Ali Akbari, first author of the study and senior staff scientist in the lab of Harvard geneticist David Reich, said in a press release.

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The data showed that genetic markers for red hair are among 479 gene variants that have been strongly favored over the past 10,000 years. One likely explanation, the researchers said, is a major shift in human history: the transition to farming.

As humans moved away from hunting and gathering and settled into agricultural societies, their environment and behavior changed radically, triggering an evolutionary "acceleration."

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While the Harvard study provides the first definitive statistical proof that red hair was actively selected during the rise of farming, the researchers noted that the exact prehistoric benefit still requires more study.

However, scientists have long pointed to vitamin D synthesis as a likely driver for the rise of these light-pigmented traits in northern climates.

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While redheads remain a minority of the global population today, the Harvard study’s analysis suggests that they may not be an evolutionary accident.

Instead, the red hair trait was "boosted" by natural selection as humans adapted to the challenges of a modern world, according to the researchers.

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The researchers urged caution in how these findings are interpreted.

"What a variant is associated with now is not necessarily why an allele propagated," the authors noted.

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Natural hormone may reduce obesity without cutting calories, scientists say

Researchers at the University of Oklahoma have discovered a hormone that appears to reverse obesity in the body.

The new study, published in the journal Cell Reports, used mouse models to identify a naturally occurring hormone called FGF21 (fibroblast growth factor 21).

The hormone "appears to work by signaling to a brain region involved in metabolism and appetite regulation, the same area targeted by the popular GLP-1 drugs," according to a university press release.

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FGF21 is reportedly already involved in the development of a drug to treat metabolic dysfunction-associated steatohepatitis (MASH), which is a form of fatty liver disease.

Lead study author Matthew Potthoff, PhD, a professor of biochemistry and physiology at OU College of Medicine and deputy director of the OU Health Harold Hamm Diabetes Center, said the study showed how the hormone sends "signals" to the lower back region of the brain.

"In our previous studies, we found that FGF21 signals to the brain instead of the liver, but we didn’t know where in the brain," he wrote.

"We thought we would find that it signaled to the hypothalamus (which is widely implicated in body weight regulation), so we were very surprised to discover that the signal was to the hindbrain, which is where the GLP-1 analogs are believed to act."

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The brain circuit behind these metabolic benefits appears to work by "mediating the effects of FGF21," Potthoff said — a process that can also be linked to side effects like gastrointestinal issues and, in some cases, bone loss.

"We hope that by identifying the specific circuit, it can help in the creation of more targeted therapies that are effective without negative side effects," he added.

The FGF21 and GLP-1 hormones act differently, although they target the same area of the brain, according to the researchers. While GLP-1 reduces food intake via appetite, FGF21 increases metabolic rate, burning energy and triggering weight loss.

Potthoff shared his hope for a new FGF21 drug that could target both weight loss and MASH, noting that additional studies are necessary to explore this potential.

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Dr. Peter Balazs MD, a hormone and weight loss specialist practicing in New York and New Jersey, said this discovery raises the possibility of targeting metabolic rate "directly," rather than relying only on calorie restriction.

"However, this is a preclinical [mouse] study using diet-induced obesity models, which is not fully relevant or similar to human obesity’s chronic metabolic adaptations," he told Fox News Digital.

FGF21 behaves differently in humans than in mice, as obese people have higher levels of the hormone in their blood, according to Balazs. This raises the question of how effective FGF21-based treatments would be.

"There are also important clinical concerns, such as whether FGF21 analogs can cause side effects like digestive issues and bone loss, which is especially risky since obesity already increases the chance of fractures," he cautioned. "The study doesn’t explain how these issues might be managed."

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Early human trials of FGF21 showed "modest weight loss" between 5% and 8%, according to Balazs, which is "less impressive" compared to GLP-1 treatments' average 15% weight loss.

It is also unclear whether the body could develop tolerance to FGF21 over time, which could reduce its effectiveness with long-term use, he added.

"The study is a nice first step toward alternative biochemical pathways for treating obesity," Balazs added. "However, clinical adoption will require safety trials that include bone density monitoring, along with confirmatory data in humans."

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Grieving mom hospitalized with rare ‘broken heart syndrome’ after veteran son’s suicide

A distraught mother who thought she was having a heart attack was instead hospitalized with broken heart syndrome — otherwise known as takotsubo syndrome (TTS) — less than a year after her veteran son tragically took his own life.

Dawn Turner, 57, of the U.K., lost her son in August of last year. 

Just last month, the mom of three awoke with "unbearable" chest pains, she said — and called an ambulance, worried she was going into cardiac arrest. But when she arrived at the hospital, doctors told her she was suffering from the effects of grief caused by a broken heart, as news agency SWNS reported. 

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TTS is a temporary, reversible heart condition often triggered by extreme emotional or physical stress, such as grief, fear or severe illness, according to experts.

Symptoms usually mimic a heart attack, with sudden and severe chest pain and shortness of breath the most common — and it primarily affects women over the age of 50.

Turner, of Eckington in Worcester, said, "I was [sitting] downstairs earlier that night and thought I had a bit of indigestion. I went to bed and just couldn’t get comfortable — I was breaking out in a sweat and had heart palpitations.

"Then, around midnight, I had pain down my arm and in my jaw. I was still putting it down to indigestion ... My partner Paul asked me if I was all right, and I said, ‘I think I'm having a heart attack.'"

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She said she couldn't catch her breath — "and my heart felt as though it was missing a beat and then [started] thudding again. For those moments, I truly believed I was having a heart attack."

She said her partner called emergency services, and an ambulance arrived within five minutes.

"They came in and linked me up to an ECG. They said, ‘Your heart is all over the place — there’s an extra beat, and it’s all over the place,’" she said, as SWNS reported. 

Turner was rushed to the hospital by ambulance.

In emergency care, Turner was also given blood tests.

She added, "They came back and said I didn’t have the enzymes produced from a heart attack in my blood. But they said there [was] something going on."

After undergoing more tests and seeing a cardiologist, Turner was told she had takotsubo syndrome.

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"I told [the doctor] that my heart feels broken. I told her about [my son] Rob, and she said it’s exactly that. She said it’s a real thing, and that I'd been under so much stress. The body can only take so much, and the grief and the stress can be quite physical."

Turner's son committed suicide in August 2025 after struggling to get help with his mental health.

He spent 10 years in the Royal Horse Artillery after joining in 2006, when he worked as an artilleryman.

He did two tours of duty in Afghanistan, she said, and returned to civilian life in 2016 before suffering several worsening health conditions.

Turner, who is also the CEO of a veterans charity called Stepway, "When he left the army, he got married, and they settled down in London. He walked straight into a job as a delivery driver. But then his health took a downward spiral, and he started having digestive troubles."

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He was eventually told he had PTSD — but those symptoms may be similar to those of mild traumatic brain injury, Turner said.

"He was deaf in one ear from using the guns," she said. "He realized he was putting so much pressure on his marriage, so he moved back up with me. He started to build himself up — then COVID hit."

Turner said there were unfortunate delays as her son tried to get access to various services and facilities.

"When people lose loved ones, you’re obviously distraught, but you eventually find closure," she said, per SWNS. "I found peace when I lost my sister in 2015. But with Rob, I can't find closure because there’s no justice there."

Turner is now on the mend and hopes to be fully recovered in a couple of weeks, SWNS reported. 

"Until that moment, I had never really understood that a person could become so overwhelmed by stress and grief that it physically affects the heart," she shared. "Broken heart syndrome can look and feel like a heart attack. It was a warning sign for me, and for anyone. It can change the shape of one of your heart chambers … it can cause some serious damage."

She added, "The cardiologist told me that thankfully, my heart itself is healthy and there was no damage, but that it will take around two weeks to a month for my heart to reboot itself."

Turner was told she needed to rest, seek counseling and make lifestyle changes to reduce stress.

"Things have settled down, and I’m taking things easy — I’m pacing myself now, and I feel a lot better. Paul said, 'Maybe the extra beat is for Rob. You are carrying on living for him.'"

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Turner said, "That broke me and healed me a little bit all at once."

Fox News Digital previously reported that broken heart syndrome, which causes the heart to temporarily weaken, has been linked to the brain’s reaction to stress, as studies have found. 

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In an article published in the European Heart Journal in March 2019, Swiss researchers said they found that the syndrome is linked to the way the brain communicates with the heart.

Caused by intense emotional events, TTS is a rare, temporary condition that weakens the left ventricle and disrupts its normal pumping function.

The syndrome causes the heart's main pumping chamber to change shape and get larger. The heart muscle becomes weaker, and its pumping action loses strength. 

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Symptoms include sudden, intense chest pain, pressure or heaviness in the chest, along with shortness of breath. 

It is treated with beta blockers and blood-thinning medicine to reduce risks of clots and other flareups.

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Common eating habit may trigger premature immune system aging, study finds

Eating too much salt has long been linked to high blood pressure, but new research suggests that it could trick the immune system into prematurely aging the blood vessels.

A preclinical study recently published in the Journal of the American Heart Association has identified a biological chain reaction that links a salty diet to cardiovascular decay.

Scientists at the University of South Alabama observed that mice on a high-salt diet experienced rapid deterioration in their blood vessel function.

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After just four weeks of high sodium intake, the small arteries responsible for regulating blood flow lost their ability to relax, as shared in a press release.

The team found that the cells lining these vessels had entered a state of cellular senescence — a form of premature cellular aging in which cells stop dividing and release a mix of inflammatory signals that can damage surrounding tissue.

The researchers tried to replicate this damage by exposing blood vessel cells directly to salt in a laboratory dish, but the cells showed no harmful effects.

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This suggests that salt isn’t directly causing damage to the vascular lining, but that the real culprit may be the body’s own defense mechanism, the researchers noted.

Excess salt may trigger the immune system to release a molecule called interleukin-16 (IL-16), which acts as a messenger that instructs blood vessel cells to grow old before their time, according to the study.

Once these cells age, they fail to produce nitric oxide, the essential gas that tells arteries to dilate and stay flexible.

To test whether this process could be reversed, the team turned to a class of experimental drugs known as senolytics.

Using a cancer medication called navitoclax, which selectively clears out aged and dysfunctional cells, the researchers were able to restore nearly normal blood vessel function in the salt-fed mice, the release stated.

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By removing the decaying cells created by the high-salt diet, the drug allowed the remaining healthy tissue to maintain its elasticity and respond correctly to blood flow demands.

The study did have some limitations. The transition from mouse models to human treatment remains a significant hurdle, the team cautioned.

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Senolytic drugs like navitoclax are still being studied for safety, and the team emphasized that previous trials have shown mixed results regarding their impact on artery plaque.

Additionally, the researchers have not yet confirmed whether the same IL-16 pathway is the primary driver of vascular aging in humans.

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More naps, higher risk? Research links daytime sleep to health warnings in aging adults

Excessive daytime napping is associated with higher mortality risk in older adults, according to new research from Mass General Brigham.

Between 20% and 60% of older adults take naps, according to the researchers.

While occasional naps have been shown to be refreshing, frequent or prolonged daytime napping in this group has been associated with a variety of health concerns.

The study followed 1,338 older adults for up to 19 years, tracking the timing and duration of their daytime napping habits using a wrist-worn tracker. They then examined all-cause mortality rates.

Taking longer, more frequent naps — or napping in the morning — were all associated with increased mortality risk and may be early warning signs of underlying health decline, as shared in a press release from Mass Brigham.

Morning naps were linked to about a 30% increase in mortality risk, and each additional hour of daily napping was associated with about a 13% higher risk, the study found. Each additional nap per day increased risk by roughly 7%.

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"Excessive napping later in life has been linked to neurodegeneration, cardiovascular diseases and even greater morbidity, but many of those findings rely on self-reported napping habits and leave out metrics like when and how regular those naps are," said lead author Chenlu Gao, PhD, an investigator in the department of anesthesiology at the Mass General Brigham, in the press release. 

"Our study is one of the first to show an association between objectively measured nap patterns and mortality, and suggests there is immense clinical value in tracking napping patterns to catch health conditions early."

There were some limitations to the study. As it was observational in design, it only showed that napping may reflect underlying illness, but did not prove that it caused mortality.

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The research did not take into account any contributing health conditions, and the sleep trackers only measured movement and not brain activity. This means rest could be misclassified as sleep in some cases.

Also, because the study population was limited to older, White adults in the Midwest, the results may not apply to other groups.

"It is important to note that this is correlation, not causation. Excessive napping is likely to indicate underlying disease, chronic conditions, sleep disturbances or circadian dysregulation," said Gao.

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"Now that we know there is a strong correlation between napping patterns and mortality rates, we can make the case to implement wearable daytime nap assessments to predict health conditions and prevent further decline."

The study was supported by grants from the National Institutes of Health and affiliated research programs.

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Psychedelic therapy may be coming to your doctor's office as questions swirl

As President Donald Trump backs efforts to advance psychedelic drugs, doctors are speaking out about how the move could impact mental health treatments.

On Friday, Trump signed an executive order to fast-track the research, funding and potential FDA approval of psychedelics like ibogaine, psilocybin, LCD and MDMA, primarily to treat PTSD, depression and addiction.

Psychedelics, psychoactive compounds that act largely through serotonin pathways, are being studied in controlled clinical settings for mental health treatment, experts say.

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Trump’s move is drawing mixed reactions, with supporters praising its potential to transform mental health treatment, particularly for veterans, while critics warn about limited evidence on safety and effectiveness.

"The president’s action today opens a pathway to research that will further open doors to expedited approval of this life-saving medicine as a treatment for our veterans — and society — who have suffered for decades from treatment-resistant PTSD, TBI (traumatic brain injury) and depression," Jay Kopelman, former Marine Corps lieutenant colonel and CEO of Mission to Live Foundation in San Diego, told Fox News Digital.

"This act will require the VA Health System to begin psychedelics research and clinical trials, making psychedelics available to veterans for whom the traditional modalities of care (SSRIs and talk therapy) haven’t worked."

Dr. Marc Siegel, Fox News senior medical analyst, agreed that psychedelics hold "big potential" for severe depression and PTSD, and said he supports Trump’s commitment to funding more research. 

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"It changes brain chemistry in a way that can provide more modulation of dopamine, serotonin and other neurochemicals, making a patient less fearful," he told Fox News Digital. 

"At the same time, the psychiatric impact of psychedelics can be useful to change perception in a positive way."

Siegel emphasized, however, that these therapies need to be further researched, and that doses and exact indications must be carefully determined.

Juliana Mercer, a 16-year Marine Corps veteran and the executive director of New York-based nonprofit dedicated to advancing psychedelic-assisted therapy for veterans, applauded the presidents' move as a "meaningful step."

"The people who need this most are those who have already tried everything and found no real relief," she told Fox News Digital.

"I think about veterans I served with, people who have done years of therapy, cycled through medications, and are still carrying the weight of their service. Too many are still losing that fight at home."

Kevin A. Sabet, Ph.D., president and CEO of the Foundation for Drug Policy Solutions in Washington, D.C., expressed concerns about the executive order.

"While we support rigorous research for treatment discovery, President Trump’s executive order on ibogaine puts politics and hype ahead of science by suggesting that a dangerous, unapproved hallucinogen can somehow be a medical treatment," he shared with Fox News Digital. 

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"Ibogaine remains a Schedule I substance with serious safety concerns, including documented cardiotoxicity and deaths. [The government] should not normalize unproven and risky drugs under the guise of helping those who have served our country."

Siegel also cautioned about the risk of improper prescribing of psychedelics by unqualified practitioners. "Unfettered recreational use and microdosing can increase the risk of psychosis and other anxiety disorders," he added.

Kopelman noted that the primary risk of ibogaine is its "cardiotoxicity properties."

"It can prolong the QT interval in the heartbeat, which can lead to arrhythmia or even heart attack," he told Fox News Digital, noting that this risk is mitigated by "rigorous medical examinations" and monitoring during treatment.

Mercer agreed that these treatments aren’t appropriate for everyone.

"They can have significant psychological effects and, in some cases, physiological risks, which is why they should only be administered in controlled clinical settings by trained providers," she told Fox News Digital.

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"Continued research is essential to better understand who these therapies are right for, and who they’re not, before broader implementation."

While Trump’s order is a step toward FDA approval for psychedelics, the substances still need to go through clinical trials and the standard regulatory pathway, including rescheduling, Mercer noted.

"Veterans and others will still be waiting unless we also build the systems to provide this safely at scale, trained providers, reimbursement pathways and clinical care models," she said. "This is the gap we’ve been highlighting for years, and it still needs to be closed."

Kopelman agreed that while the legislation paves a path for expedited study of these medications, it’s important to "do it the right way," warning that "one misstep could overturn and sink this entire effort."

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"Most important is the post-medicine integration work that requires multiple therapy sessions overseen by a trauma-informed psychedelic therapist," he told Fox News Digital. "It’s not like prescribing someone a Tylenol and sending them home — these medicines require medical oversight."

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"Psychedelics are not a panacea," Kopelman added. "They are a gateway to healing — but the real work begins after treatment."

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Healthy diets spark lung cancer risk in non-smokers as pesticides loom

Eating a diet high in fruits and vegetables was found to have a surprising link to lung cancer among younger non-smokers, early research suggests.

The observational study, led by Jorge Nieva, MD, of the USC Norris Comprehensive Cancer Center at Keck Medicine, was presented this month at the American Association for Cancer Research (AACR) annual meeting in San Diego. It has not yet been peer-reviewed. 

Researchers looked at dietary, smoking and demographic data for 187 patients who were diagnosed with lung cancer at age 50 or younger. 

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They found that among non-smokers, there was a link between healthier-than-average diets – rich in fruits, vegetables and whole grains – and the chance of lung cancer development.

Young lung cancer patients ate more servings of dark green vegetables, legumes and whole grains compared to the average U.S. adult, the researchers found.

The researchers hypothesized that pesticides applied to conventionally grown produce could be a possible factor in the disease association.

"Commercially produced (non-organic) fruits, vegetables and whole grains are more likely to be associated with a higher residue of pesticides than dairy, meat and many processed foods," according to Nieva. He also noted that agricultural workers exposed to pesticides tend to have higher rates of lung cancer.

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"There is a large subset of lung cancer patients whose disease is not caused by smoking," Nieva told Fox News Digital.

The disease is becoming more common in non-smokers 50 and younger, especially women – despite the fact that smoking rates have been falling for decades, the researcher noted.

"These patients tend to have eaten much healthier diets before their diagnosis than the average American," he went on. "We need to support research into understanding why Americans – and women in particular – who no longer smoke very much are still having lung cancer," he said.

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The study did have some limitations, Nieva acknowledged, primarily that it relied on survey data and was limited by the participants’ memories of their food intake.  

"Also, the survey participants were self-selected, and this could have biased the findings," he told Fox News Digital.

The researchers did not test specific foods for pesticides, relying instead on average pesticide levels for certain types of food. Looking ahead, they plan to test patients’ blood and urine samples to directly measure pesticide levels, Nieva said.

Although the study shows only an association and does not prove that pesticides caused lung cancer, Nieva recommends that people wash their produce before eating and choose organic foods whenever possible.

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"This work represents a critical step toward identifying modifiable environmental factors that may contribute to lung cancer in young adults," said Nieva. "Our hope is that these insights can guide both public health recommendations and future investigation into lung cancer prevention."  

Dr. Marc Siegel, Fox News senior medical analyst, said the study is "interesting," but that it "raises far more questions than it answers."

"It is a small study (around 150) and observational, so no proof," the doctor, who was not involved in the research, told Fox News Digital.

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"It is possible that the increased lung cancer risk could be due to pesticide exposure in whole farmed foods, but it is by no means certain," Siegel went on. "How much exposure is needed? How much of it gets into food and in which areas? This requires much further study."

Kayla Nichols, communications director for Pesticide Action & Agroecology Network, a distributed global network, said the organization agrees with the study's conclusion that more research should be done on the rise in lung cancer, particularly in individuals eating diets higher in produce and fiber.

"There is a bounty of existing research that already links pesticide exposure to increased risk of multiple types of cancers," Nichols, who was also not involved in the study, told Fox News Digital. She called for more research on chronic, low-level exposures to pesticides, as well as more effective policies to protect the public from pesticide residues on food.

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The study was supported by the National Institutes of Health and the National Cancer Institute, as well as industry partners including AstraZeneca and Genentech, among others.

Fox News Digital reached out to several pesticide companies and trade groups for comment.

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